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doi: 184

Among the module membership was GCL, composed of the catalytic GCLC and the modifier GCLM subunit, shown to be upregulated (Fig

Henceforth, inhibition of RACK1 and MDH2 genes was found to cause up-regulation of SCN1A genes

Subsequently, the signal is transferred to collapsin response mediator protein 2 (CRMP2) that regulates cytoskeletal organization and thereby axonal outgrowth/repulsion