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10.1016/j.freeradbiomed.2008.10.022 105 RafiyanM.SadeghmousaviS.AkbarzadehmoallemkolaeiM.RezaeiN

As a research tool without commercial pharmaceutical development, this investment has not been made

Targeting codon 158 p53-mutant cancers via the induction of p53 acetylation

S15, n = 10 per group) was performed because recurrence is clinically associated with survival outcomes

Torsemide versus furosemide after discharge in patients hospitalized with heart failure across the spectrum of ejection fraction: findings from TRANSFORM-HF

These axes interact with the central nervous system through distinct molecular mechanisms and play significant roles in the initiation, propagation, and progression of PD