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There is a separate type of steatosis by which phospholipid accumulation leads to a pattern similar to the diseases with inherited phospholipid metabolism defects (e.g., TaySachs disease) Causes: (a) Microvesicular: Aspirin (Reye's syndrome), ketoprofen, tetracycline (especially if expired) (b) Macrovesicular: Acetaminophen, methotrexate (c) Phospholipidosis: Amiodarone, total parenteral nutrition (d) Antiviral: nucleoside analogues (e) Corticosteroid (f) Hormonal: Tamoxifen Granuloma [edit] Drug-induced hepatic granulomas are usually associated with granulomas in other tissues and patients typically have features of systemic vasculitis and hypersensitivity

Implication of intestinal barrier dysfunction in gut dysbiosis and diseases

(2) a non-amyloid-component (NAC)

Short-chain fatty acids induce acute phosphorylation of the p38 mitogen-activated protein kinase/heat shock protein 27 pathway via GPR43 in the MCF-7 human breast cancer cell line

In this experiment, GSH content and GCL activity in gills were reduced after hypoxia stress, suggesting that hypoxia May inhibit glutathione biosynthesis, whereas supplementation with 291.90 to 583.80 mg/kg glutathione elevated GSH levels and GCL activity, thereby alleviating hypoxia-induced glutathione depletion

Antioxid Basel Switz 2023, 12:834