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Description

Mitochondrial lipids in neurodegeneration

Cysteine availability can be targeted through its systemic depletion 2 or inhibition of the primary Cys 2 transporter, system X c 6

Low dose dimethyl sulfoxide driven gross molecular changes have the potential to interfere with various cellular processes

Transcriptomics reveals changes in gene expression, while metabolomics reflects alterations in cellular metabolic states

Validation using shotgun metagenomic sequencing or metabolomic analyses will be necessary to confirm whether these predicted pathway differences translate into actual metabolic alterations in CHD