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Yet, established clinical markers for AA remain elusive, emphasizing the need for further research in diagnostic and therapeutic modalities

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In this mechanism, the GSH-dependent cystine reduction pathway stimulates the production of GSH by reducing the cysteine that system Xc transports into the cell [33,34,35]

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GSH activity is facilitated by activation of the Kelch-like ECH-associated protein 1 (Keap1)-Nuclear factor erythroid 2-related factor 2 (Nrf2)-antioxidant response element (ARE) redox regulator pathway, releasing Nrf2 that regulates expression of genes controlling antioxidant, inflammatory and immune system responses