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Notably, in this study, vitamin D appears to influence the development of pulmonary fibrosis through multiple biological pathways: vitamin D deficiency not only exacerbates the lung inflammation caused by bleomycin (similar to the effect observed in the case of vitamin C deficiency), but also increases the likelihood of the occurrence of EMT in the lungs, and these are all crucial steps in the pathogenesis of pulmonary fibrosis

Fudenberg and coworkers addressed this question from a different viewpoint and asked whether heat-eluted IgG from senescent RBC has the same specificity as anti-Thomson-Friedenreich antibodies (anti-T), that are generated against neuraminidase-treated RBC glycoproteins

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Toxic iron species in lower-risk myelodysplastic syndrome patients: course of disease and effects on outcome

Soil acidity: development, impacts, and management