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If so, would it be better to apply a double punch approach by alternating drugs targeting different cancer cell features, rather than exploiting pulses of the same anticancer agent, or would constant exposure to lethal doses of a therapeutic regimen exert a stronger pressure on DTP plasticity, thus resulting in a prolonged effect

This approach mirrors dosing strategies used for other peptides with 1216 hour half-lives, though published 5-amino-1mq pharmacokinetic data in humans remains limited

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