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Overall, activation of these signaling pathways, which are tightly linked to cellular stress responses, promotes the release of pro-inflammatory mediators and cytokines from both airway structural and immune cells, thereby establishing a self-perpetuating inflammatory loop that sustains and aggravates asthma pathology

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While all six agents increased GPX4 levels, melatonin, LBP, PAH, FA, and EGCG were also found to increase SCL7A11 levels, and TFERL, LBP, PAH, FA, and EGCG also increased levels of GSH [38, 48, 92,93,94,95]