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(2018) ( PMID: 30069438

Therefore, the implication from this work is that a large portion of the S in normal cellular steady-state GSH pools is derived from Met via the Met-cycle and transsulfuration

47:W212W224

Intestine-specific FTN-deficiency leads to marked increases in intestinal iron absorption and inducing iron overload, and an unidentified novel microbial metabolite regulates systemic FTN homeostasis (Das et al., 2020)

The high biocompatibility of exosomes provides clinicians with more opportunities to create safe targeted therapies for the treatment and management of IBD and CRC