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Principal component analysis (PCA), metabolic pathway enrichment analysis, and Venn diagram generation were accomplished using the Origin 2019 software (OriginLab Corporation, Northampton, MA)

PMID: 25843792

The fact that Yap1 deficiency extends the egtA phenotype indicates that in wild-type, EGT limitation is partially compensated by other detoxification mechanisms that require activation by Yap1

Mutations in GBA and LRRK2 further impair mitophagy signaling, compounding mitochondrial dysfunction and accelerating disease progression