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More specifically, orforglipron is a potent partial GLP-1 RA which is biased towards G-protein activation over to -arrestin recruitment at the GLP-1 receptor [23]

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Notably, enforced expression of KEAP1 in Cis_NR T24 and J82 cells sensitized them to cisplatin in a dose-dependent manner, further confirming the critical role of KEAP1 in mediating chemosensitivity (Supplementary Fig

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The activated state correlated with elevated transcription of the pro-inflammatory cytokine IL-1, indicating that MP exposure triggers microglial activation and subsequent hypersecretion of inflammatory mediators, thereby accelerating neuronal damage 31