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10.1002/ddr.70044 Summary Keywords cisplatin-induced nephrotoxicity, ferroptosis, LicoA, mitochondrion, Nrf2 Citation Han M, Zhang F, Li M, Zhang Q, Shen K, Zhang D, Xu H, Zhou F and Wang X (2026) Licochalcone A mitigates cisplatin-induced nephrotoxicity by inhibiting ferroptosis and mitochondrial dysfunction via the Nrf2/GPX4 axis

The conversion of GSSG back to GSH is accomplished by the enzyme glutathione reductase (GR) (Figure 1)

Splitting the dose or adding a second daily injection is not part of the clinical protocol and may not improve outcomes

While our bodies naturally produce this powerful tripeptide, levels of glutathione can deplete due to stress, poor diet, aging, and other lifestyle factors

The Boc-protected cystine-1,1- 13 C was reacted with two equivalents of glycine tert -butyl ester (OtBu) hydrochloride using 1-ethyl-3-(3-dimethylaminopropyl)carbodiimide monohydrochloride and 1-hydroxybenzotriazole to produce the N and C-protected dipeptidyl (Boc-Cys-Gly-OtBu) disulfide derivative

Many experience nutrient deficiencies due to restricted eating patterns, sensory sensitivities, gastrointestinal issues, or chronic stress