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As used herein, the term treatment of skin damage means the treatment of the symptoms of skin damage, treatment of the skin to prevent or reduce the damage from reactive carbonyl species (RCS), glycation of skin proteins, formation of advanced glycation endproducts (AGEs) and formation of advanced lipoxidation endproducts(ALEs) and/or to prevent or reduce the further accumulation of reactive carbonyl species (RCS), glycated skin proteins, advanced glycation endproducts (AGEs) and advanced lipoxidation endproducts(ALEs) in the skin

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TNX appears to be fibril-associated and is localized in the spaces between fibrils by immunology electron microscopy of bovine and human skin

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