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On the contrary, it acts as a compulsory exchanger, mediating the influx of leucine into the cell and the efflux of glutamine out of the cell [29, 42]

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Detection of ProteinProtein Interactions Using Glutathione-S-Transferase (GST) Pull-Down Assay Technique

The escalating combinations: High CYP1B1 activity + null GSTM1/GSTT1: CYP1B1 Leu432Val high-activity variants increase 4-OHE2 production from estradiol providing more reactive substrate for quinone formation

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These results indicate that NINJ1 regulates ferroptosis specifically induced by xCT inhibition