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As -KG enters the TCA cycle, it gets oxidized to generate succinate and fumarate, which are cancer metabolites that produce ATP, NADH, and FADH2 [34] (Fig

Such modulation is essential to mitigate the harmful consequences of reperfusion injury since it can be used to sustain cellular homeostasis and avoid a high inflammatory state (Yousefi Zardak et al., 2024)

However, despite the growing consensus on their clinical relevance, the precise definition and identification of CSCs remain subjects of ongoing debate

Oxidative stress as a second line of attack occurs after the onset of initial pathology, documented, for example, by the formation of superoxide radicals and hydrogen peroxide by NADPH oxidases, for example, in the response to injury-mediated inflammation

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