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A large-scale review published in the journal of Nutrition & Metabolism looked for possible mechanisms by which glutathione levels could affect autism risk

This characteristic has been implicated in the intrinsic resistance of NTHi to siderophore-targeting compounds observed in vivo

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When acetaminophen is consumed in toxic doses, the pathways leading to nontoxic metabolites are overwhelmed and the accumulation of NAPQI depletes glutathione

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Many of these biological effects have been noted with microorganisms (Li et al., 2010) in wastewater treatment plants (Kiser et al., 2010), the natural environment, and the human digestive system (Zhang et al., 2009)