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As cited above, the amino acid cysteine is a rate-limiting factor for GSH synthesis, and a variety of both clinical trials and in-vitro/in-vivo data suggest that supplying cysteine as NAC is an effective strategy for enhancing GSH production and intracellular cysteine.Intravenous NAC has been utilized for some time to treat acetaminophen and non-acetaminophen induced acute liver failure by restoring the concentration of GSH, and has been found to be safe and effective

Traditional therapies primarily rely on RAAS blockers, such as ACE inhibitors or ARBs, which can slow albuminuria progression but offer limited efficacy in late-stage DN and fail to reverse renal fibrosis (Samsu, 2021)

doi: 10.1093/bioinformatics/btn628 83 KadyrbaevM

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Pfeifer H, Conrad M, Roethlein D, Kyriakopoulos A, Brielmeier M, Bornkamm GW et al

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