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Both in vitro and in vivo studies have revealed that abnormalities in the mitochondrial complex may disrupt the redox equilibrium, and that a reduction in Co Q10 concentration may increase free radicals without scavenging them, thereby inducing neuronal degeneration (Mischley et al., 2016)

It has also been shown to interact with the NO system, providing endothelial protection and angiogenic effects even in severely impaired conditions

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