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[47] evaluated the effects of parenteral administration of the alanyl-glutamine dipeptide in total parenteral nutrition associated with liver damage in infant rabbits and observed that this dipeptide supplementation can minimize liver damage and reduce malondialdehyde (MDA) production and hepatocyte apoptosis during total parenteral nutrition

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Cochrane Database Syst Rev 2009 Oct 7;(4):CD006884

Inhibiting HDAC1 lactylation at K412 facilitates the significant activation of FTO and ALKBH5, which reduces ferroptosis suppressor protein ( FSP1 ) mRNA m 6 A methylation in CRC cells