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By including pharmacological antagonists targeting GHSR, CCR5, and CXCR4, the researchers also aimed to clarify the specific receptor-mediated mechanisms through which GHRP-6 influences muscle pathology and recovery [2]

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While current pharmacological treatments provide symptomatic relief and modestly slow disease progression, they fail to address the underlying pathophysiology and are often accompanied by severe adverse effects

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471,472 For instance, acetate can regulate inflammatory responses by affecting lipid accumulation in macrophages in a dose-dependent manner