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glutathione is likely to be oxidised by the burst of ROS released from over-reduced electron transport components and metabolic NADP reduction can re-commence due to oxygen-dependent metabolic flux through the relevant enzyme systems such as those of the cytosolic oxidative pentose phosphate pathway

I., Londono, M., Hirschberger, L

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The accumulation of harmful metabolites and depletion of protective metabolites contribute to fibrosis progression through various mediators, including the renin-angiotensin system, reactive oxygen species, Toll-like receptor 4, aryl hydrocarbon receptor, inhibitor of kappa B/nuclear factor kappa B, and Kelch-like ECH-associated protein 1/nuclear factor erythroid 2-related factor 2 pathways

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