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Advances in Glutathione Research for Disease Research Targeted therapy for GSH metabolism has become a research hotspot, and metabolomics and proteomics technologies have also provided powerful tools for GSH-related research: Targeting GSH metabolism : Inhibiting key molecules such as GCLC, xCT (cysteine uptake carrier) or SLC25A39 (mitochondrial GSH transporter), or using GSH-depleting nanozymes, has shown the potential to enhance ROS-mediated tumor cell death and improve related disease symptoms in preclinical models (such as cell models and animal models), providing new strategies for disease treatment

Na(+)/I(-) symporter and Pendred syndrome gene and protein expressions in human extra-thyroidal tissues

The Maillard reaction products as food-born antioxidant and antibrowning agents in model and real food systems

NAD+: Enhances mitochondrial function, reduces oxidative stress, and supports cartilage integrity

For example, hyperhomocysteinemia (linked to gut microbial metabolism) can alter DNA methylation in the brain and has been associated with increased AD pathology [114]

Common neurological manifestations include: Paraesthesia (pins and needles) in the hands and feet Numbness and altered sensation Difficulty with balance and coordination (ataxia) Memory problems and cognitive impairment Mood changes, including depression and irritability In severe cases, visual disturbances and confusion These neurological features arise from demyelination of peripheral nerves and the spinal cord, a condition known as subacute combined degeneration when it affects the posterior and lateral columns of the spinal cord