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Kaempferol via the Syk-PLC and PKC-ERK-cPLA2-COX2 and NF-B signaling pathways ameliorates airway hyperplasia and hypertrophy (Gong et al

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Another possibility would be a higher stability and resistance to proteolytic degradation of hybrid fibrils, shifting the equilibrium away from the toxic oligomers toward the less toxic fibrils

on the other hand, perhaps enhanced ER retention time and chaperone association of client proteins facilitates proper protein folding under stressful conditions