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As shown above, the NHEJ repair activity was observed to be around 5.4% and 4.7% for transfection by T3-CRISPR/Cas9-PEI-MNPs magnetoplexes with and without the influence of magnetic field

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In parallel, the mTORC1 inhibitor rapamycin increased LC3 expression in normal fibroblasts, as assessed by western blotting and immunofluorescence analyses (Fig

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saturatedation mutagenesis includes a method that uses degenerate oligonucleotide primers to introduce point mutations into a polynucleotide, as described in detail, below