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Mitochondrial dysfunction from any cause (biotoxin illness, heavy metals, oxidative stress) directly impairs amino acid conjugation because the activation step requires mitochondrial ATP and CoA

H3K27ac and H3K4me1 are associated with active enhancers [19], and high levels of H3K4me3 and H3 and H4 acetylation are found at the promoters of active genes [20, 21]

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Therefore, whether KIR2DS4 can be considered a druggable gene for cataracts requires further investigation in more appropriate experimental models

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