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Djavaheri-Mergny M, Javelaud D, Wietzerbin J, Besanon F (2004) NF-kappaB activation prevents apoptotic oxidative stress via an increase of both thioredoxin and MnSOD levels in TNFalpha-treated Ewing sarcoma cells

In chronic wounds, persistent inflammation is often due to a failure in the M1-to-M2 macrophage transition, driven by excessive pro-inflammatory cytokine production and inefficient clearance of apoptotic neutrophils [33]

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T cell self-reactivity forms a cytokine milieu for spontaneous development of IL-17+ Th cells that cause autoimmune arthritis

Either way, the goal is the same, keep them stable for their intended use