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However, in the study conducted by Huiya Li et al., cell debris generated through the repeated freezethaw cycles of HT22 cell lines in the OGD/R model inadequately represented the substantial quantity of myelin debris produced following ischemic injury

In this model, inflammation drives a massive upregulation of proangiogenic factors, which overwhelms the antiangiogenic mechanisms of the cornea and results in a secondary ingrowth of both blood and lymphatic vessels from the limbal area into the corneal center 15

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