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and Wells, W

Brain iron accumulation exacerbates the pathogenesis of MPTP-induced Parkinsons disease

An orally-available selective nonpeptide SST5 agonist, CRN02481, has been shown to suppress insulin secretion and increase glucose levels in both oral glucose tolerance tests and a sulfonylurea model of hyperinsulinism ( -/- mouse model of CHI ( Conclusions The biology of SST receptors and ability of SRLs to activate specific receptor subtypes to reduce excess insulin is important in the understanding of the pathobiology and treatment perspectives in CHI

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BPC-157 is not FDA-approved for human use and currently appears on the FDAs Category 2 list of compounds restricted from routine compounding

AMBRA1 is able to induce mitophagy via LC3 binding, regardless of PARKIN and p62/SQSTM1