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Nevertheless, it is worth noting that cationic substances exhibit a high affinity for anionic cell membranes found in all living cells 6

Recent work indicates that ATII cells play a pivotal role in the comorbid pathogenesis of IPF and lung cancer-associated fibrosis ( The NLRP3 inflammasome, a critical intracellular multiprotein complex of innate immunity, can be activated by danger signals such as silica (SiO 2 )

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Furthermore, research has shown that autophagy-mediated iron death plays a significant role in IPF, with bisphenol A (BPA) enhancing autophagy-mediated ferroptosis through activation of the AMPK/mTOR signaling pathway, thereby exacerbating the progression of IPF [116]