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Despite the inherent resistance of EMS tissue to ferroptosis, studies have shown that Erastin can still effectively induce ferroptosis in EMS lesions, leading to tissue atrophy and cell death

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Because BPC-157 is associated with signaling pathways that influence vascular formation and collagen-associated cellular responses, while TB-500 influences actin-driven cellular movement, the combination allows researchers to explore how structural tissue remodeling may be coordinated at multiple biological levels [1,5,6]

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