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A key player in the ferroptosis pathway is nuclear erythroid 2-related factor 2 (NRF2), a transcription factor that controls the expression of many antioxidant genes and components of ferroptosis [75]

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Brain iron chelation by deferiprone in a phase 2 randomised double-blinded placebo controlled clinical trial in parkinsons disease

The reason behind that has been speculated to be due the fact that the occurrence of monoallelic IDH1 and IDH2 mutations is more frequent by chance than the biallelic mutations expected for IDH3 [44]

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Looking at one study, even in fairly sensitive asthmatic patients only 4 of 24 had a reaction to extremely high sulfite levels (45mg dose from 300mg/L concentration)