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SODs efficiently catalyze the dismutation of highly reactive superoxide, which is primarily generated within the mitochondrial matrix as a product of single electron reduction of molecular oxygen through the electron transport chain (Miao and St Clair 2009

Mechanisms of NADPH Oxidase Activation: Translocation of P40phox, Rac1 and Rac2 from the Cytosol to the Membranes in Human Neutrophils Lacking P47phox or P67phox

These strategies have generated encouraging preclinical results, particularly in improving bioavailability, reducing peripheral adverse effects, and enhancing therapeutic retention within the central nervous system