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Enhancing fusion in pancreatic adenocarcinoma models, either via mitofusin 2 (MFN2) overexpression or DRP1 inhibition, paradoxically increased mitophagy, leading to reduced mitochondrial mass and OXPHOS capacity

Inappropriate p53 activation during development induces features of CHARGE syndrome

In the context of limited glutamine, various cell components in the TME compete for glutamine, and tumor cells can increase their uptake of glutamine due to their unique metabolic mode, thus providing certain competitive advantages

Furthermore, the novel classification was validated in an independent cohort of 34 samples from Beijing

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