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Thus, this preliminary evidence proposes that structured KD protocols can safely and effectively modulate host metabolism during advanced cancer treatment

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Crucially, this pathway is regulated by mTORC2, suggesting that tumor cells with deregulated mTOR signaling may possess unique mechanisms to either exploit or suppress this lytic release of antigens [80]

In one aspect, the method comprises identifying an antigen binding site variant by its increased antigen binding affinity or antigen binding specificity as compared to the affinity or specificity of the first antigen binding site to the antigen