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acetylation, methylation or glycosylation), transduce the information on the cell metabolic status, and modulate the activities of signaling proteins, enzymes, and transcriptional regulators 3

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Dose-dependent associations existed between L-carnitine levels and risk of coronary artery disease, peripheral artery disease, and CVD but only in participants with concurrently high TMAO levels [3][7][36]

The interaction of phage and biofilms

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A proof of the dynamic balancing of carnitine homeostasis derives from the observation that, under regular diet, patients carrying defects of the carnitine biosynthesis enzymes, do not apparently display carnitine deficiency, because the reduced synthesis is compensated by carnitine dietary intake and increased renal reabsorption (El-Hattab and Scaglia, 2015)