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Within mitochondria, covalent binding of NAPQI to nucleophilic proteins disrupts the respiratory chain, enhances reactive oxygen species generation, dissipates mitochondrial membrane potential, and ultimately triggers opening of the mitochondrial permeability transition (MPT) pore, leading to necrotic cell death (Ramachandran and Jaeschke 2019b)

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This disruption of Wnt signaling creates a permissive environment for CSC differentiation and loss of tumor-initiating potential

Without adequate levels, the liver may struggle to process these compounds efficiently

The treated groups showed improvement in hepatic tissue, with nearly regular hepatic cords and sinusoids

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