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Consequently, expression of IL-2, TNF-, and IFN- was reduced, leading to impaired anti-tumor function ( 2+ influx and excessive activation of NFAT ( Conversely, studies examining the effects of methionine in the context of immune checkpoint blockade (ICB) therapies indicate a more suppressive role

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Sigma-2 receptor antagonists rescue neuronal dysfunction induced by Parkinsons patient brain-derived -synuclein

Increasing evidence indicates that both conditions are associated with immune activation and pro-inflammatory states (Maes et al., 2008)

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