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MIF null mice are entirely resistant to the detrimental effects of OVX, implying that MIF acts as a key downstream mediator of both the detrimental effects of estrogen deficiency and the beneficial effects of estrogen replacement on skin wound healing [57, 175, 182]

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This indicates the potential role of GABA not only as a metabolite but also as a signaling molecule, influencing metabolic adjustments in response to necrotrophs (Barbosa et al

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This is especially true for those who are severely deficient