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however, its uptake by the neuronal tissue should be strictly regulated to prevent toxicity (Linert and Kozlowski, 2012

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Along these lines, an emerging theory linking excitotoxicity and mitochondrial dysfunction suggests that an accumulation of lactate, a metabolic byproduct which is toxic (especially to nerve cells) at high concentrations may play a role in ALS progression (Vadakkadath Meethal 2012)

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An endotoxin-free peptide helps ensure that observed effects are attributable to Cagrilintide itself rather than external contaminants