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In turn, enhancing KCC2 activity not only potentiated L838,417-induced analgesia, it rescued its analgesic potential at high doses, revealing a novel strategy for analgesia in pathological pain, by combined targeting of the appropriate GABA A R-subtypes and restoring Cl - homeostasis

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Previous study established a LPS-induced murine sepsis model ( in vitro experiments demonstrated that MMPP inhibited cardiomyocyte death by attenuating oxidative stress and inflammatory responses while maintaining mitochondrial homeostasis

Together, these observations suggest that inflammatory cytokine signalling can act as a common axis linking neural dysfunction and tumor progression through context-dependent modulation of Wnt and JAK/STAT pathways

The histopathological features of SJS/TEN are extensive necrosis of epidermal keratinocytes, mainly mediated by drug-specific cytotoxic T cells [18]

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