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doi: 10.1016/j.molcatb.2009.09.010

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Here, such difficulties are overcome through re-bridging of the inter-chain disulfides of cetuximab (CTX) with auristatin-bearing pyridazinediones, to yield a highly refined anti-epidermal growth factor receptor (EGFR) ADC

Pharmacological activation of AMP-activated protein kinase (AMPK) inhibits Glut1 expression, thereby promoting fatty acid oxidation and increasing FoxP3 expression, which is a lineage marker for Treg cells [101]