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Emerging preclinical data suggest that electronic nicotine delivery system exposure may induce ferroptosis-associated lung injury through increased CD71/TFR1 and ACSL4 expression, reduced GPX4, altered lipid profiles, BODIPY-C11-positive lipid peroxidation, epithelial cell death, inflammation, mucus accumulation, emphysema-like pathology, and fibrosis

Meanwhile, single-target strategies may provoke unwanted immune responses in the TME in certain cases, necessitating the further optimization of combination therapies

Following similar trends, uric acid concentration, which acts as an antioxidant 38,39 , also decreased with range use in the strain S757N

Nutr Neurosci 24:697709

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