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Neurotoxicol Teratol

Clinical and Experimental Hepatology 6(2):106115

Excitotoxic stimuli can damage oligodendroglial cells by means of Ca 2+ entry which leads to Ca 2+ dyshomeostasis and mitochondrial membrane alterations

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Even though the underlying pathways that mediate the development of tubulointerstitial lesions in diabetic nephropathy are not well understood, previous studies have suggested that TGF-1 is the principal mediator of the deleterious effects of the diabetic milieu 11