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however, evidence is emerging that increased cellular GSSG accumulation may be of equal, if not greater, importance in regulating oxidative stress-triggered detrimental effects

In: Rosenthal GA, Janzen DH, editors

MASLD serves as an umbrella term that encompasses a broad spectrum of pathological liver changes from simple hepatic steatosis to steatohepatitis associated with metabolic dysfunction, which can lead to fibrosis and eventually progress to cirrhosis or hepatocellular carcinoma (HCC), while simultaneously increasing the risk of cardiovascular diseases (CVDs) and extrahepatic malignancies [1]

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Each monomer has a binding site for guanosine triphosphate (GTP) and another for nicotinamide adenine diphosphate hydride (NADH)/adenosine diphosphate (ADP)