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Conclusion Collectively, we unveil GPR68 as a critical sensor of ischemic acidosis that orchestrates ferroptosis resistance via PI3K/Akt/Nrf2 signaling

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Eukaryotes lack this capacity and in particular complex animals can excrete absorbed Mercury only with difficulty, therefore most of the dose accumulates over time in long-half-life forms, from which the metal can be released and reach biochemically-impairing levels and thus exert toxic effects

Quan X, Xu X, Guo G, Ou Y, Shang H, Wei Y

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