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Mouse models of anemia of inflammatory disease (and hemolytic anemia) have shown that monocytes transiently convert into erythrophagocytic macrophages in the liver (Theurl et al 2016)

This interaction modulates the equilibrium among Th1, Th2, Th17, Treg, and other T cell subsets, subsequently regulating inflammatory responses ( + T cell production of IL-2, correcting abnormal expression of IL-17A and IL-2, and benefiting CD4 + T cells in lupus prone mice and SLE patients ( Figure 3 )

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