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doi: 10.5483/BMBRep.2023-0098 Summary Keywords acute pancreatitis, mitochondrial, calcium overload, mitochondrial permeability transition pore, regulated cell death Citation Chen F, Xu K, Han Y, Ding J, Ren J, Wang Y, Ma Z and Cao F (2024) Mitochondrial dysfunction in pancreatic acinar cells: mechanisms and therapeutic strategies in acute pancreatitis

Results indicated that although telomere length and hTERT were elevated after treatment with epitalon, actual telomerase activity did not follow the same pattern

75 These findings reinforce the notion that, rather than initiating dopaminergic cell death, inflammation may perpetuate it, thus contributing to Parkinson's disease progression

Th1 and Th2 cells: different patterns of lymphokine secretion lead to different functional properties

In autoantibody-treated mice, C-fibers had lower mechanical thresholds and higher firing frequency and total action potentials during a 0- to 100-mN force ramp stimulus, with increased firing at relatively low forces (Fig