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The treatment of GSH results in increased enzymatic activities such as SOD, CAT, GPX, GR, MDHAR, and DHAR, which leads to an increase in endogenous AsA and GSH levels, along with increased ratios of AsA/DHA and GSH/GSSG, thus reducing the level of overproduced ROS species (Jung et al., 2019

However, various neurodevelopmental disorder conditions, including ASD, may cause steadily high levels of NO in the brain, promoting abnormal SNO of mitochondrial proteins