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Despite promising preclinical findings, clinical evidence supporting astaxanthins neuroprotective effects remains limited

Most patients self-administer in under a minute

Its main mechanism of action would be its ability to bind to serum calcium, preventing activation of the coagulation cascade and prolonging the time till blood clot formation

Turnbull, J

Although compensatory mechanisms attempt to counteract these harmful effects, prolonged astrocyte activation often leads to irreversible pathological conditions, tipping the balance toward neuronal degeneration (Fig

In summary, PF results suggest that PF could regulate mood disorders and exert antidepressant-like effects by modulating the disordered HPA axis function