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Studies have shown that mitochondrial therapeutics may have the potential to slow the progression of POAG by protecting against mitochondrial decline associated with age, genetic susceptibility, and other pathology

Interestingly, while antagonism of YIL781 and TAK-779 did not interfere with this effect, the administration of AMD3100, a CXCR4 antagonist, markedly reduced the LC3 II-to-LC3 I ratio in DOX-treated muscle co-administered with GHRP-6, suggesting that CXCR4 signaling plays a crucial role in mediating autophagic responses [2]

Ion channels in osteoarthritis: emerging roles and potential targets

2017;12(12):e0189310

doi: 10.1300/J092v11n01_02 15 CarruthersBMvan de SandeMIDe MeirleirKLKlimasNGBroderickGMitchellTet al

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